Seroprevalance of toxocara antibodies in multiple sclerosis and ankylosing spondylitis.

The other is a humoral hypothesis, which proposes a immune-mediated, inflammatory response, with release of vasoactive substances capable of producing endothelial damage and alterations of permeability.[4] In this case, it is unlikely to have been due to a demyelinating process, as the MRI Brain and the CSF examination was normal. This case had focal neurological deficits of hemiparesis and cerebellar dysfunction, which resolved with adequate treatment of malaria. This case is being reported for its rarity and to highlight the role of early recognition and timely treatment, which leads to complete recovery.